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The influence of oral fluid volume on the intestinal absorption of a variety of agents was examined in four dogs using various measures of drug transit. Simultaneous collection of oral and duodenal fluid was utilized to monitor the drug and water entry into the intestinal lumen. Whole gut transit time (Tg) and duodenal fluid excretion rates (QD) were measured by the method of Neville. The Jellinek calculus was modified to quantify the amount of drug absorbed at any given time of transit. Comparison of drug and water transit through the alimentary tract demonstrated a distinct lumen-mucosa transit sequence with time. The bulk of drug transport was accomplished in three out of four cases, as measured by both Tg and QD. In the fourth dog, however, there was a lag of as much as five hours between the appearance of drug and fluid in the duodenum and the commencement of drug absorption, as reflected by Tg. Conversely, the Jellinek calculus was insensitive to the observed lag between drug and fluid absorption as reflected by Tg. Thus, in the presence of a significant lag time, QD and Tg may not be appropriate for the accurate prediction of intestinal drug transport.The long-term objective of this study is to further our understanding of the molecular mechanisms underlying the lack of B7 costimulation signals in the human immune system. We hypothesize that dysregulation of B7 signaling molecules in humans may be the cause for autoimmune diseases, like autoimmune thyroiditis and celiac disease. Our specific aims are: (1) to determine if dominant negative TGF- binding proteins (TGF-BP) can block TGF- action, and to find the exact cause(s) for the functional difference(s) between the two major classes of TGF-BP, activin-binding protein (ACTHBP) and endoglin (ENG); and (2) to determine the role of TGF-BP in the etiology of celiac disease. If human TGF-BP can block TGF- action in human cells, as we have observed in mouse cells, then the pharmacological utility of TGF-BP as a clinical

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